Research output per year
Research output per year
Personal profile
Personal profile
L. Pablo Cid Soto is a physician graduated from the University of Chile. After working as a general practitioner, he had an academic position at the Faculty of Medicine of the University of Chile in the Department of Experimental Medicine. He did postdoctoral training at the Department of Biophysics and Physiology at Johns-Hopkins University (Baltimore, USA). Since 2000 he has been a researcher at the Center for Scientific Studies (CECs, Valdivia)
Teaching
He has done undergraduate teaching activities in Pathophysiology at the Faculty of Medicine of the University of Chile and postgraduate in the PhD program in Medical Sciences and PhD program in Sciences mention Molecular and Cellular Biology from the Austral University of Chile
Research interests
His main interest has been to investigate the role of potassium and chloride ion channels in physiology and pathophysiology at cellular as well as systemic level using genetically modified mouse models. He has also been interested in the study of glutamate receptors in the copepod Caligus rogercresseyi, especially their role in resistance to the antiparasitic ivermectin
Expertise related to UN Sustainable Development Goals
In 2015, UN member states agreed to 17 global Sustainable Development Goals (SDGs) to end poverty, protect the planet and ensure prosperity for all. This person’s work contributes towards the following SDG(s):
Education/Academic qualification
Master, Ciencias Médicas mención en Fisiopatología, Universidad de Chile
… → 2000
Bachelor, Médico cirujano, Universidad de Chile
… → 1983
Keywords
- QP Physiology
Cargo USS
- Investigador (a)
Jerarquía Académica
- Profesor Asociado
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Collaborations and top research areas from the last five years
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A new family of glutamate-gated chloride channels in parasitic sea louse Caligus rogercresseyi: A subunit refractory to activation by ivermectin is dominant in heteromeric assemblies
Tribiños, F., Cuevas, P., Cornejo, I., Sepúlveda, F. V. & Pablo Cid, L., 2023, In: PLoS Pathogens. 19, 3 March, p. e1011188 e1011188.Research output: Contribution to journal › Article › peer-review
Open Access1 Scopus citations -
Altered phosphatidylinositol regulation of mutant inwardly rectifying K+ Kir7.1 channels associated with inherited retinal degeneration disease
Vera, E., Cornejo, I., Niemeyer, M. I., Sepúlveda, F. V. & Cid, L. P., 2021, In: Journal of Physiology. 599, 2, p. 593-608 16 p.Research output: Contribution to journal › Article › peer-review
Open Access2 Scopus citations -
Short Chain Fatty Acids Effect on Chloride Channel ClC-2 as a Possible Mechanism for Lubiprostone Intestinal Action
Catalán, M. A., Julio-Kalajzić, F., Niemeyer, M. I., Cid, L. P. & Sepúlveda, F. V., 2020, In: Cells. 9, 8, 210.Research output: Contribution to journal › Article › peer-review
Open Access5 Scopus citations -
A novel Kir7.1 splice variant expressed in various mouse tissues shares organisational and functional properties with human Leber amaurosis-causing mutations of this K+ channel
Vera, E., Cornejo, I., Burgos, J., Niemeyer, M. I., Sepúlveda, F. V. & Cid, L. P., 2019, In: Biochemical and Biophysical Research Communications. 514, 3, p. 574-579 6 p.Research output: Contribution to journal › Article › peer-review
7 Scopus citations -
Kir7.1 inwardly rectifying K+ channel is expressed in ciliary body non pigment epithelial cells and might contribute to intraocular pressure regulation
Burgos, J., Villanueva, S., Ojeda, M., Cornejo, I., Cid, L. P. & Sepúlveda, F. V., 2019, In: Experimental Eye Research. 186, 107723.Research output: Contribution to journal › Article › peer-review
4 Scopus citations