A novel Kir7.1 splice variant expressed in various mouse tissues shares organisational and functional properties with human Leber amaurosis-causing mutations of this K+ channel
Research output: Contribution to journal › Article › peer-review
7Scopus
citations
Fingerprint
Dive into the research topics of 'A novel Kir7.1 splice variant expressed in various mouse tissues shares organisational and functional properties with human Leber amaurosis-causing mutations of this K+ channel'. Together they form a unique fingerprint.