Personal profile
Personal profile
L. Pablo Cid Soto is a physician graduated from the University of Chile. After working as a general practitioner, he had an academic position at the Faculty of Medicine of the University of Chile in the Department of Experimental Medicine. He did postdoctoral training at the Department of Biophysics and Physiology at Johns-Hopkins University (Baltimore, USA). Since 2000 he has been a researcher at the Center for Scientific Studies (CECs, Valdivia)
Teaching
He has done undergraduate teaching activities in Pathophysiology at the Faculty of Medicine of the University of Chile and postgraduate in the PhD program in Medical Sciences and PhD program in Sciences mention Molecular and Cellular Biology from the Austral University of Chile
Research interests
His main interest has been to investigate the role of potassium and chloride ion channels in physiology and pathophysiology at cellular as well as systemic level using genetically modified mouse models. He has also been interested in the study of glutamate receptors in the copepod Caligus rogercresseyi, especially their role in resistance to the antiparasitic ivermectin
Education/Academic qualification
Master, Ciencias Médicas mención en Fisiopatología, Universidad de Chile
… → 2000
Bachelor, Médico cirujano, Universidad de Chile
… → 1983
Keywords
- QP Physiology
Cargo USS
- Investigador (a)
Jerarquía Académica
- Profesor Asociado
Expertise related to UN Sustainable Development Goals
In 2015, UN member states agreed to 17 global Sustainable Development Goals (SDGs) to end poverty, protect the planet and ensure prosperity for all. This person’s work contributes towards the following SDG(s):
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SDG 3 Good Health and Well-being -
SDG 6 Clean Water and Sanitation -
SDG 13 Climate Action -
SDG 14 Life Below Water
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Collaborations and top research areas from the last five years
Recent external collaboration on country/territory level. Dive into details by clicking on the dots or
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Normal vision and development in mice with low functional expression of Kir7.1 in heterozygosis for a blindness-producing mutation inactivating the channel: NA
Cornejo Araneda, I. M., Sepúlveda Valenzuela, F. L., Cid Soto, L. P., Vera, E., Henao, J. C., Tribiños, F. & Burgos, J., 2024, In: American Journal of Physiology - Cell Physiology. 326, 4, p. C1178-C1192Research output: Contribution to journal › Article › peer-review
4 Scopus citations -
A new family of glutamate-gated chloride channels in parasitic sea louse Caligus rogercresseyi: A subunit refractory to activation by ivermectin is dominant in heteromeric assemblies
Tribiños, F., Cuevas, P., Cornejo, I., Sepúlveda, F. V. & Pablo Cid, L., 2023, In: PLoS Pathogens. 19, 3 March, e1011188.Research output: Contribution to journal › Article › peer-review
Open Access5 Scopus citations -
Altered phosphatidylinositol regulation of mutant inwardly rectifying K+ Kir7.1 channels associated with inherited retinal degeneration disease
Vera, E., Cornejo, I., Niemeyer, M. I., Sepúlveda, F. V. & Cid, L. P., 2021, In: Journal of Physiology. 599, 2, p. 593-608 16 p.Research output: Contribution to journal › Article › peer-review
5 Scopus citations -
Short Chain Fatty Acids Effect on Chloride Channel ClC-2 as a Possible Mechanism for Lubiprostone Intestinal Action
Catalán, M. A., Julio-Kalajzić, F., Niemeyer, M. I., Cid, L. P. & Sepúlveda, F. V., 2020, In: Cells. 9, 8, 210.Research output: Contribution to journal › Article › peer-review
Open Access7 Scopus citations -
A novel Kir7.1 splice variant expressed in various mouse tissues shares organisational and functional properties with human Leber amaurosis-causing mutations of this K+ channel
Vera, E., Cornejo, I., Burgos, J., Niemeyer, M. I., Sepúlveda, F. V. & Cid, L. P., 2019, In: Biochemical and Biophysical Research Communications. 514, 3, p. 574-579 6 p.Research output: Contribution to journal › Article › peer-review
8 Scopus citations